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Januar 2008 / Adv Genet Channelopathies of transepithelial transport and vesicular function C.A. Huebner T.J. Jentsch 01. Oktober 2000 / Nat Rev Neurosci Neuronal KCNQ potassium channels: physiology and role in disease T.J. Jentsch 16. November 2000 / Nature ClC-5 Cl- -channel disruption impairs endocytosis in a mouse model for Dent's disease N. Piwon W. Guenther M. Schwake M.R. Boesl T.J. Jentsch 01. August 2000 / Epilepsia Pathophysiology of KCNQ channels: neonatal epilepsy and progressive deafness T.J. Jentsch B.C. Schroeder C. Kubisch T. Friedrich V. Stein 12. August 2000 / Hum Mol Genet Mutations in the a3 subunit of the vacuolar H(+)-ATPase cause infantile malignant osteopetrosis U. Kornak A. Schulz W. Friedrich S. Uhlhaas B. Kremens T. Voit C. Hasan U. Bode T.J. Jentsch C. Kubisch 01. August 2000 / J Am Soc Nephrol Characterization of renal chloride channel (CLCN5) mutations in Dent's disease K. Yamamoto J.P. Cox T. Friedrich P.T. Christie M. Bald P.N. Houtman M.J. Lapsley L. Patzer M. Tsimaratos W.G. Van'T Hoff K. Yamaoka T.J. Jentsch R.V. Thakker 01. Juli 2000 / J Am Soc Nephrol From tonus to tonicity: physiology of CLC chloride channels S. Waldegger T.J. Jentsch 01. Juni 2000 / Curr Opin Neurobiol Neurological diseases caused by ion-channel mutations F. Weinreich T.J. Jentsch 11. August 2000 / J Biol Chem Functional and structural analysis of ClC-K chloride channels involved in renal disease S. Waldegger T.J. Jentsch 04. August 2000 / J Biol Chem KCNQ5, a novel potassium channel broadly expressed in brain, mediates M-type currents B.C. Schroeder M. Hechenberger F. Weinreich C. Kubisch T.J. Jentsch Seitennummerierung Aktuelle Seite 1 Seite 2 Seite 3 Seite 4 … Nächste Seite Next › Letzte Seite Last »
01. Januar 2008 / Adv Genet Channelopathies of transepithelial transport and vesicular function C.A. Huebner T.J. Jentsch
01. Oktober 2000 / Nat Rev Neurosci Neuronal KCNQ potassium channels: physiology and role in disease T.J. Jentsch
16. November 2000 / Nature ClC-5 Cl- -channel disruption impairs endocytosis in a mouse model for Dent's disease N. Piwon W. Guenther M. Schwake M.R. Boesl T.J. Jentsch
01. August 2000 / Epilepsia Pathophysiology of KCNQ channels: neonatal epilepsy and progressive deafness T.J. Jentsch B.C. Schroeder C. Kubisch T. Friedrich V. Stein
12. August 2000 / Hum Mol Genet Mutations in the a3 subunit of the vacuolar H(+)-ATPase cause infantile malignant osteopetrosis U. Kornak A. Schulz W. Friedrich S. Uhlhaas B. Kremens T. Voit C. Hasan U. Bode T.J. Jentsch C. Kubisch
01. August 2000 / J Am Soc Nephrol Characterization of renal chloride channel (CLCN5) mutations in Dent's disease K. Yamamoto J.P. Cox T. Friedrich P.T. Christie M. Bald P.N. Houtman M.J. Lapsley L. Patzer M. Tsimaratos W.G. Van'T Hoff K. Yamaoka T.J. Jentsch R.V. Thakker
01. Juli 2000 / J Am Soc Nephrol From tonus to tonicity: physiology of CLC chloride channels S. Waldegger T.J. Jentsch
01. Juni 2000 / Curr Opin Neurobiol Neurological diseases caused by ion-channel mutations F. Weinreich T.J. Jentsch
11. August 2000 / J Biol Chem Functional and structural analysis of ClC-K chloride channels involved in renal disease S. Waldegger T.J. Jentsch
04. August 2000 / J Biol Chem KCNQ5, a novel potassium channel broadly expressed in brain, mediates M-type currents B.C. Schroeder M. Hechenberger F. Weinreich C. Kubisch T.J. Jentsch