Wissenschaftliche Publikationen Suche Suche Autor/in Forschungsgruppe Veröffentlichungsdatum Wirkungsfaktor Suchen Sortieren nach RelevanceFrom A-ZAuthored onDate/Time RangeRelease date Reihenfolge AufsteigendAbsteigend Morano, Ingo Prof. Dr. (1) Rathjen, Fritz Prof. Dr. (1) Wellner, Maren Dr. (1) Willnow, Thomas Prof. Dr. (2) (-) Wanker, Erich Prof. Dr. (17) Biobank (4) Entwicklungsbiologie / Signaltransduktion in Nerven und Muskelzellen (1) Experimentelle Ultrahochfeld-MR (3) Integrative Vaskuläre Biologie (9) Magnetic Resonance (3) Molekulare Epidemiologie (4) Molekulare Immunologie und Gentherapie (3) Molekulare Physiologie der somatosensorischen Wahrnehmung (14) Myologie (5) (-) Proteomforschung und molekulare Mechanismen bei neurodegenerativen Erkrankungen (17) 1991 (1) 1995 (4) (-) 1997 (4) 1998 (7) (-) 1999 (7) 2000 (5) 2001 (6) 2002 (5) (-) 2003 (6) 2004 (7) 2005 (8) 2006 (13) 2007 (6) 2008 (6) 2009 (6) 2010 (4) 2011 (6) 2012 (9) 2013 (11) 2014 (9) 2015 (8) 2016 (10) 2017 (6) 2018 (10) 2019 (6) 2020 (11) 2021 (6) 2022 (3) 2023 (5) 2024 (2) 17 Ergebnisse: Active Filter: Wanker, Erich Prof. Dr.Proteomforschung und molekulare Mechanismen bei neurodegenerativen Erkrankungen199719992003 Sortieren: Treffgenauigkeit Neueste nach älteste Älteste nach neueste 01. August 2003 / Trends Biochem Sci The hunt for huntingtin function: interaction partners tell many different stories P. Harjes E.E. Wanker 17. Oktober 2003 / J Biol Chem Mutant huntingtin promotes the fibrillogenesis of wild-type huntingtin - A potential mechanism for loss of huntingtin function in Huntington's disease A. Busch S. Engemann R. Lurz H. Okazawa H. Lehrach E.E. Wanker 01. Januar 2003 / J Biol Chem Lithostathine quadruple-helical filaments form proteinase K-resistant deposits in Creutzfeldt-Jakob disease E. Laurine C. Gregoire M. Faendrich S. Engemann S. Marchal L. Thion M. Mohr B. Monsarrat B. Michel C.M. Dobson E.E. Wanker M. Erard J.M. Verdier 01. Januar 2003 / J Neurochem Histone deacetylase activity is retained in primary neurons expressing mutant huntingtin protein M. Hoshino K. Tagawa T. Okuda M. Murata K. Oyanagi N. Arai T. Mizutani I. Kanazawa E.E. Wanker H. Okazawa 08. August 1997 / Cell Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation S.W. Davies M. Turmaine B.A. Cozens M. DiFiglia A.H. Sharp C.A. Ross E. Scherzinger E.E. Wanker L. Mangiarini G.P. Bates 01. Januar 1999 / Methods Enzymol Detection of polyglutamine aggregation in mouse models S.W. Davies K. Sathasivam C. Hobbs P. Doherty L. Mangiarini E. Scherzinger E.E. Wanker G.P. Bates 29. Juni 1999 / Philos Transact A Math Phys Eng Sci Aggregation of truncated GST-HD exon 1 fusion proteins containing normal range and expanded glutamine repeats B. Hollenbach E. Scherzinger K. Schweiger R. Lurz H. Lehrach E.E. Wanker 01. Mai 1999 / Hum Mol Genet Formation of polyglutamine inclusions in non-CNS tissue K. Sathasivam C. Hobbs M. Turmaine L. Mangiarini A. Mahal F. Bertaux E.E. Wanker P. Doherty S.W. Davies G.P. Bates 08. August 1997 / Cell Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo E. Scherzinger R. Lurz M. Turmaine L. Mangiarini B. Hollenbach R. Hasenbank G.P. Bates S.W. Davies H. Lehrach E.E. Wanker 13. April 1999 / Proc Natl Acad Sci U S A Self-assembly of polyglutamine-containing huntingtin fragments into amyloid-like fibrils: implications for Huntington's disease pathology E. Scherzinger A. Sittler K. Schweiger V. Heiser R. Lurz R. Hasenbank G.P. Bates H. Lehrach E.E. Wanker Seitennummerierung Aktuelle Seite 1 Seite 2 Nächste Seite Next › Letzte Seite Last »
01. August 2003 / Trends Biochem Sci The hunt for huntingtin function: interaction partners tell many different stories P. Harjes E.E. Wanker
17. Oktober 2003 / J Biol Chem Mutant huntingtin promotes the fibrillogenesis of wild-type huntingtin - A potential mechanism for loss of huntingtin function in Huntington's disease A. Busch S. Engemann R. Lurz H. Okazawa H. Lehrach E.E. Wanker
01. Januar 2003 / J Biol Chem Lithostathine quadruple-helical filaments form proteinase K-resistant deposits in Creutzfeldt-Jakob disease E. Laurine C. Gregoire M. Faendrich S. Engemann S. Marchal L. Thion M. Mohr B. Monsarrat B. Michel C.M. Dobson E.E. Wanker M. Erard J.M. Verdier
01. Januar 2003 / J Neurochem Histone deacetylase activity is retained in primary neurons expressing mutant huntingtin protein M. Hoshino K. Tagawa T. Okuda M. Murata K. Oyanagi N. Arai T. Mizutani I. Kanazawa E.E. Wanker H. Okazawa
08. August 1997 / Cell Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation S.W. Davies M. Turmaine B.A. Cozens M. DiFiglia A.H. Sharp C.A. Ross E. Scherzinger E.E. Wanker L. Mangiarini G.P. Bates
01. Januar 1999 / Methods Enzymol Detection of polyglutamine aggregation in mouse models S.W. Davies K. Sathasivam C. Hobbs P. Doherty L. Mangiarini E. Scherzinger E.E. Wanker G.P. Bates
29. Juni 1999 / Philos Transact A Math Phys Eng Sci Aggregation of truncated GST-HD exon 1 fusion proteins containing normal range and expanded glutamine repeats B. Hollenbach E. Scherzinger K. Schweiger R. Lurz H. Lehrach E.E. Wanker
01. Mai 1999 / Hum Mol Genet Formation of polyglutamine inclusions in non-CNS tissue K. Sathasivam C. Hobbs M. Turmaine L. Mangiarini A. Mahal F. Bertaux E.E. Wanker P. Doherty S.W. Davies G.P. Bates
08. August 1997 / Cell Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo E. Scherzinger R. Lurz M. Turmaine L. Mangiarini B. Hollenbach R. Hasenbank G.P. Bates S.W. Davies H. Lehrach E.E. Wanker
13. April 1999 / Proc Natl Acad Sci U S A Self-assembly of polyglutamine-containing huntingtin fragments into amyloid-like fibrils: implications for Huntington's disease pathology E. Scherzinger A. Sittler K. Schweiger V. Heiser R. Lurz R. Hasenbank G.P. Bates H. Lehrach E.E. Wanker