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in Exp Physiol

Angiotensin‐II receptor type Ia does not contribute to cardiac atrophy following high‐thoracic spinal cord injury in mice

  • A. Järve
  • F. Qadri
  • M. Todiras
  • S. Schmolke
  • M. Bader
in Front Physiol

Angiotensin-(1-7) receptor Mas deficiency does not exacerbate cardiac atrophy following high-level spinal cord injury in mice

  • A. Järve
  • F. Qadri
  • M. Todiras
  • S. Schmolke
  • N. Alenina
  • M. Bader
in Exp Neurol

Distinct roles of angiotensin receptors in autonomic dysreflexia following high-level spinal cord injury in mice

  • A. Järve
  • M. Todiras
  • X Lian
  • R. Filippelli-Silva
  • F. Qadri
  • R.P. Martin
  • M. Gollasch
  • M. Bader
in Front Physiol

Angiotensin-(1-7) receptor Mas in hemodynamic and thermoregulatory dysfunction after high-level spinal cord injury in mice: a pilot study

  • A. Järve
  • M. Todiras
  • M. Kny
  • F.I. Fischer
  • J.F. Kraemer
  • N. Wessel
  • R. Plehm
  • J. Fielitz
  • N. Alenina
  • M. Bader
in FASEB J

Adverse left ventricular remodeling by glycoprotein nonmetastatic melanoma protein B in myocardial infarction

  • A. Järve
  • S. Mühlstedt
  • F. Qadri
  • B. Nickl
  • H. Schulz
  • N. Hübner
  • C. Özcelik
  • M. Bader
in J Mol Med

Cardiomyocyte-derived CXCL12 is not involved in cardiogenesis but plays a crucial role in myocardial infarction

  • S. Mühlstedt
  • S.K. Ghadge
  • J. Duchene
  • F. Qadri
  • A. Järve
  • L. Vilianovich
  • E. Popova
  • A. Pohlmann
  • T. Niendorf
  • P. Boyé
  • C. Özcelik
  • M. Bader