Wissenschaftliche Publikationen Suche Suche Autor/in Forschungsgruppe Veröffentlichungsdatum Suchen Sortieren nach RelevanceFrom A-ZAuthored onDate/Time RangeRelease date Reihenfolge AufsteigendAbsteigend Kammertöns, Thomas Dr. (2) Kieback, Elisa Dr. (1) Kopp, Joachim Dr. (1) Leisegang, Matthias Prof. Dr. rer. nat. (1) Pezzutto, Antonio Prof. Dr. (1) Schlag, Peter M. Prof. Dr. (1) Uckert, Wolfgang Prof. Dr. (3) Willimsky, Gerald Dr. (3) (-) Blankenstein, Thomas Prof. Dr. (10) (-) Daniel, Peter Prof. Dr. (1) Biologie maligner Lymphome (6) Intrazelluläre Proteolyse (1) Mathematische Modellierung zellulärer Prozesse (1) (-) Molekulare Immunologie und Gentherapie (10) Proteom Dynamik (10) RNA Biologie und Posttranscriptionale Regulation (1) Systembiologie von Gen-regulatorischen Elementen (2) Translationale Onkologie solider Tumore (1) 1992 (4) 1993 (7) 1994 (5) 1995 (8) 1996 (6) 1997 (9) 1998 (9) 1999 (2) 2000 (8) 2001 (5) 2002 (5) 2003 (9) 2004 (8) 2006 (1) 2007 (5) (-) 2008 (5) 2009 (2) 2010 (7) (-) 2011 (5) 2012 (6) 2013 (11) 2014 (3) 2015 (4) 2016 (4) 2017 (6) 2018 (4) 2019 (6) 2021 (4) 2022 (6) 2023 (4) 2024 (2) 10 Ergebnisse: Active Filter: Blankenstein, Thomas Prof. Dr.Daniel, Peter Prof. Dr.Molekulare Immunologie und Gentherapie 20082011 Sortieren: Treffgenauigkeit Neueste nach älteste Älteste nach neueste 07. Juli 2008 / J Exp Med Immunogenicity of premalignant lesions is the primary cause of general cytotoxic T lymphocyte unresponsiveness G. Willimsky M. Czeh C. Loddenkemper J. Gellermann K. Schmidt P. Wust H. Stein T. Blankenstein 01. Oktober 2008 / Int J Cancer SV40 large T antigen-transformed human primary normal and cancerous mammary epithelial cells are phenotypically similar but can be distinguished in 3D culture with selection medium L.P. Li G. Willimsky S. Seitz Y. Xu Y. Li L.E. Schwarz P.M. Schlag T. Blankenstein 08. September 2008 / Canc Lett Ablation of TNF or lymphotoxin signaling and the frequency of spontaneous tumors in p53-deficient mice D.V. Kuprash Z. Qin D. Ito S.I. Grivennikov K. Abe L.N. Drutskaya T. Blankenstein S.A. Nedospasov 15. Januar 2008 / Proc Natl Acad Sci U S A A safeguard eliminates T cell receptor gene-modified autoreactive T cells after adoptive transfer E. Kieback J. Charo D. Sommermeyer T. Blankenstein W. Uckert 01. November 2011 / Eur J Immunol Visualizing the dynamic of adoptively transferred T cells during the rejection of large established tumors J. Charo C. Perez C. Buschow A. Jukica M. Czeh T. Blankenstein 28. Juli 2011 / Blood The only proposed T-cell epitope derived from the TEL-AML1 translocation is not naturally processed J. Popovic L.P. Li P.M. Kloetzel M. Leisegang W. Uckert T. Blankenstein 11. November 2008 / Proc Natl Acad Sci U S A Engineering antigen-specific primary human NK cells against HER-2 positive carcinomas A. Kruschinski A. Moosmann I. Poschke H. Norell M. Chmielewski B. Seliger R. Kiessling T. Blankenstein H. Abken J. Charo 01. April 2011 / Gene Ther Allogeneic gene-modified tumor cells (RCC-26/IL-7/CD80) as a vaccine in patients with metastatic renal cell cancer: a clinical phase-I study J. Westermann A. Floercken G. Willimsky A. van Lessen J. Kopp A. Takvorian K. Joehrens A. Lukowsky C. Schoenemann B. Sawitzki H. Pohla R. Frank B. Doerken D.J. Schendel T. Blankenstein A. Pezzutto 15. Januar 2011 / Int J Cancer Tumor rejection by local interferon gamma induction in established tumors is associated with blood vessel destruction and necrosis D. Briesemeister D. Sommermeyer C. Loddenkemper R. Loew W. Uckert T. Blankenstein T. Kammertoens 13. Dezember 2011 / Canc Cell Oncogene-targeting T cells reject large tumors while oncogene inactivation selects escape variants in mouse models of cancer K. Anders C. Buschow A. Herrmann A. Milojkovic C. Loddenkemper T. Kammertoens P. Daniel H. Yu J. Charo T. Blankenstein
07. Juli 2008 / J Exp Med Immunogenicity of premalignant lesions is the primary cause of general cytotoxic T lymphocyte unresponsiveness G. Willimsky M. Czeh C. Loddenkemper J. Gellermann K. Schmidt P. Wust H. Stein T. Blankenstein
01. Oktober 2008 / Int J Cancer SV40 large T antigen-transformed human primary normal and cancerous mammary epithelial cells are phenotypically similar but can be distinguished in 3D culture with selection medium L.P. Li G. Willimsky S. Seitz Y. Xu Y. Li L.E. Schwarz P.M. Schlag T. Blankenstein
08. September 2008 / Canc Lett Ablation of TNF or lymphotoxin signaling and the frequency of spontaneous tumors in p53-deficient mice D.V. Kuprash Z. Qin D. Ito S.I. Grivennikov K. Abe L.N. Drutskaya T. Blankenstein S.A. Nedospasov
15. Januar 2008 / Proc Natl Acad Sci U S A A safeguard eliminates T cell receptor gene-modified autoreactive T cells after adoptive transfer E. Kieback J. Charo D. Sommermeyer T. Blankenstein W. Uckert
01. November 2011 / Eur J Immunol Visualizing the dynamic of adoptively transferred T cells during the rejection of large established tumors J. Charo C. Perez C. Buschow A. Jukica M. Czeh T. Blankenstein
28. Juli 2011 / Blood The only proposed T-cell epitope derived from the TEL-AML1 translocation is not naturally processed J. Popovic L.P. Li P.M. Kloetzel M. Leisegang W. Uckert T. Blankenstein
11. November 2008 / Proc Natl Acad Sci U S A Engineering antigen-specific primary human NK cells against HER-2 positive carcinomas A. Kruschinski A. Moosmann I. Poschke H. Norell M. Chmielewski B. Seliger R. Kiessling T. Blankenstein H. Abken J. Charo
01. April 2011 / Gene Ther Allogeneic gene-modified tumor cells (RCC-26/IL-7/CD80) as a vaccine in patients with metastatic renal cell cancer: a clinical phase-I study J. Westermann A. Floercken G. Willimsky A. van Lessen J. Kopp A. Takvorian K. Joehrens A. Lukowsky C. Schoenemann B. Sawitzki H. Pohla R. Frank B. Doerken D.J. Schendel T. Blankenstein A. Pezzutto
15. Januar 2011 / Int J Cancer Tumor rejection by local interferon gamma induction in established tumors is associated with blood vessel destruction and necrosis D. Briesemeister D. Sommermeyer C. Loddenkemper R. Loew W. Uckert T. Blankenstein T. Kammertoens
13. Dezember 2011 / Canc Cell Oncogene-targeting T cells reject large tumors while oncogene inactivation selects escape variants in mouse models of cancer K. Anders C. Buschow A. Herrmann A. Milojkovic C. Loddenkemper T. Kammertoens P. Daniel H. Yu J. Charo T. Blankenstein