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Prof. Dr.Waiczies, Sonia PD Dr.199520002012 Sortieren: Treffgenauigkeit Neueste nach älteste Älteste nach neueste 01. August 2000 / J Am Soc Nephrol Characterization of renal chloride channel (CLCN5) mutations in Dent's disease K. Yamamoto J.P. Cox T. Friedrich P.T. Christie M. Bald P.N. Houtman M.J. Lapsley L. Patzer M. Tsimaratos W.G. Van'T Hoff K. Yamaoka T.J. Jentsch R.V. Thakker 01. Juli 2000 / J Am Soc Nephrol From tonus to tonicity: physiology of CLC chloride channels S. Waldegger T.J. Jentsch 01. Juni 2000 / Curr Opin Neurobiol Neurological diseases caused by ion-channel mutations F. Weinreich T.J. Jentsch 11. August 2000 / J Biol Chem Functional and structural analysis of ClC-K chloride channels involved in renal disease S. Waldegger T.J. Jentsch 04. August 2000 / J Biol Chem KCNQ5, a novel potassium channel broadly expressed in brain, mediates M-type currents B.C. Schroeder M. Hechenberger F. Weinreich C. Kubisch T.J. Jentsch 11. April 2000 / Proc Natl Acad Sci U S A KCNQ4, a K+ channel mutated in a form of dominant deafness, is expressed in the inner ear and the central auditory pathway T. Kharkovets J.P. Hardelin S. Safieddine M. Schweizer A. El-Amraoui C. Petit T.J. Jentsch 01. Februar 2000 / J Physiol Inhibition of KCNQ1-4 potassium channels expressed in mammalian cells via M1 muscarinic acetylcholine receptors A.A. Selyanko J.K. Hadley I.C. Wood F.C. Abogadie T.J. Jentsch D.A. Brown 13. Januar 2000 / Nature A constitutively open potassium channel formed by KCNQ1 and KCNE3 B.C. Schroeder S. Waldegger S. Fehr M. Bleich R. Warth R. Greger T.J. Jentsch 29. Dezember 1995 / J Biol Chem Cloning and functional expression of rat CLC-5, a chloride channel related to kidney disease K. Steinmeyer B. Schwappach M. Bens A. Vandewalle T.J. Jentsch 11. Dezember 1995 / FEBS Lett ClC-6 and ClC-7 are two novel broadly expressed members of the CLC chloride channel family S. Brandt T.J. 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01. August 2000 / J Am Soc Nephrol Characterization of renal chloride channel (CLCN5) mutations in Dent's disease K. Yamamoto J.P. Cox T. Friedrich P.T. Christie M. Bald P.N. Houtman M.J. Lapsley L. Patzer M. Tsimaratos W.G. Van'T Hoff K. Yamaoka T.J. Jentsch R.V. Thakker
01. Juli 2000 / J Am Soc Nephrol From tonus to tonicity: physiology of CLC chloride channels S. Waldegger T.J. Jentsch
01. Juni 2000 / Curr Opin Neurobiol Neurological diseases caused by ion-channel mutations F. Weinreich T.J. Jentsch
11. August 2000 / J Biol Chem Functional and structural analysis of ClC-K chloride channels involved in renal disease S. Waldegger T.J. Jentsch
04. August 2000 / J Biol Chem KCNQ5, a novel potassium channel broadly expressed in brain, mediates M-type currents B.C. Schroeder M. Hechenberger F. Weinreich C. Kubisch T.J. Jentsch
11. April 2000 / Proc Natl Acad Sci U S A KCNQ4, a K+ channel mutated in a form of dominant deafness, is expressed in the inner ear and the central auditory pathway T. Kharkovets J.P. Hardelin S. Safieddine M. Schweizer A. El-Amraoui C. Petit T.J. Jentsch
01. Februar 2000 / J Physiol Inhibition of KCNQ1-4 potassium channels expressed in mammalian cells via M1 muscarinic acetylcholine receptors A.A. Selyanko J.K. Hadley I.C. Wood F.C. Abogadie T.J. Jentsch D.A. Brown
13. Januar 2000 / Nature A constitutively open potassium channel formed by KCNQ1 and KCNE3 B.C. Schroeder S. Waldegger S. Fehr M. Bleich R. Warth R. Greger T.J. Jentsch
29. Dezember 1995 / J Biol Chem Cloning and functional expression of rat CLC-5, a chloride channel related to kidney disease K. Steinmeyer B. Schwappach M. Bens A. Vandewalle T.J. Jentsch
11. Dezember 1995 / FEBS Lett ClC-6 and ClC-7 are two novel broadly expressed members of the CLC chloride channel family S. Brandt T.J. Jentsch